Annual Update in Intensive Care and Emergency Medicine 2012 by W. F. Carson IV, S. L. Kunkel (auth.), Prof. Jean-Louis

By W. F. Carson IV, S. L. Kunkel (auth.), Prof. Jean-Louis Vincent (eds.)

The Yearbook compiles the latest advancements in experimental and scientific study and perform in a single entire reference ebook. The chapters are written via good well-known specialists within the box of in depth care and emergency drugs. it's addressed to every body focused on inner medication, anesthesia, surgical procedure, pediatrics, in depth care and emergency medicine.

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Intensive Care Med 36: 1859 – 1866 4. Benjamim CF, Hogaboam CM, Kunkel SL (2004) The chronic consequences of severe sepsis. J Leukoc Biol 75: 408 – 412 5. Tinsley KW, Grayson MH, Swanson PE, et al (2003) Sepsis induces apoptosis and profound depletion of splenic interdigitating and follicular dendritic cells. J Immunol 171: 909 – 914 6. Wen H, Dou Y, Hogaboam CM, Kunkel SL (2008) Epigenetic regulation of dendritic cellderived interleukin-12 facilitates immunosuppression after a severe innate immune response.

Kumar V, Sharma A (2010) Is neuroimmunomodulation a future therapeutic approach for sepsis? Int Immunopharmacol 10: 9 – 17 7. Madden K, Sanders V, Felten D (1995) Catecholamine influences and sympathetic neural modulation of immune responsiveness. Annu Rev Pharmacol Toxicol 35: 417 – 448 The Neuroendocrine Axis: The Nervous System and Inflammation 8. Weihe E, Nohr D, Michel S, et al (1991) Molecular anatomy of the neuro-immune connection. Int J Neurosci 59: 1 – 23 9. John C, Buckingham J (2003) Cytokines: Regulation of hypothalamo-pituitary-adrenocortical axis.

Of particular interest is the effect of MLL haploinsufficiency on the function of bone marrow-derived macrophages (BMDM), as MLL+/- BMDM exhibit increased phagocytosis in vitro as compared to BMDM from littermate control animals (MLL+/-) (Fig. 1b). The specific role of MLL in governing phagocytosis remains to be elucidated; however, these preliminary results suggest that inhibition of MLL function may be a promising therapeutic target for sepsis. Much work remains to be done concerning the role of histone modifying enzymes in mediating sepsis and sepsis-induced immunosuppression.

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